Ver Investigador - - Prisma - Unidad de Bibliometría

Cristina Porras Gonzalez

Tipo Año Título Fuente
Artículo2024 Tissue‐specific early and late activated lymphocytes immunophenotype in chronic rhinosinusitis with nasal polyps International Forum of Allergy and Rhinology
Artículo2023 Role of fibroblasts in chronic inflammatory signalling in chronic rhinosinusitis with nasal polyps-a systematic review. JOURNAL OF CLINICAL MEDICINE
Artículo2023 Tissue-specific activated regulatory lymphocytes immunophenotype in chronic rhinosinusitis with nasal polyps ARCHIVOS DE BRONCONEUMOLOGIA
Artículo2023 Transcriptional analysis of nasal polyps fibroblasts reveals a new source of pro-inflammatory signaling in CRSwNP. Rhinology
Artículo2022 Usefulness of bilateral mucoplasty plus reboot surgery in severe type-2 chronic rhinosinusitis with nasal polyps Rhinology
Artículo2019 PKCα-mediated downregulation of RhoA activity in depolarized vascular smooth muscle: Synergistic vasorelaxant effect of PKCα and ROCK inhibition CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
Artículo2018 Contribution of L-type Ca2+ channel-sarcoplasmic reticulum coupling to depolarization-induced arterial contraction in spontaneously hypertensive rats HYPERTENSION RESEARCH
Artículo2017 Regulation of RhoA/ROCK and sustained arterial contraction by low cytosolic Ca2+ levels during prolonged depolarization of arterial smooth muscle VASCULAR PHARMACOLOGY
Artículo2015 Role of L-type Ca2+ channels, sarcoplasmic reticulum and Rho kinase in rat basilar artery contractile properties in a new model of subarachnoid hemorrhage VASCULAR PHARMACOLOGY
Resumen congreso2014 A new functional role of Ca2+sensitization mechanisms in the regulation of vascular smooth muscle contraction ACTA PHYSIOLOGICA
Artículo2014 Contractile responses to rat urotensin II in resting and depolarized basilar arteries JOURNAL OF PHYSIOLOGY AND BIOCHEMISTRY
Artículo2014 Low-dose combination of Rho kinase and L-type Ca2+ channel antagonists for selective inhibition of depolarization-induced sustained arterial contraction EUROPEAN JOURNAL OF PHARMACOLOGY
Revisión2013 A new metabotropic role for L-type Ca2+ channels in vascular smooth muscle contraction CURRENT VASCULAR PHARMACOLOGY
Artículo2012 Tonic arterial contraction mediated by L-type Ca2+ channels requires sustained Ca2+ influx, G protein-associated Ca2+ release, and RhoA/ROCK activation EUROPEAN JOURNAL OF PHARMACOLOGY
Artículo2012 Tungstate activates BK channels in a beta subunit- and Mg2+-dependent manner: relevance for arterial vasodilatation CARDIOVASCULAR RESEARCH
Artículo2011 Metabotropic Regulation of RhoA/Rho-Associated Kinase by L-type Ca2+ Channels New Mechanism for Depolarization-Evoked Mammalian Arterial Contraction CIRCULATION RESEARCH
Resumen congreso2011 Voltage-dependent regulation of intracellular calcium release in vascular smooth muscle JOURNAL OF VASCULAR RESEARCH
Artículo2010 Genetic Ablation of L-Type Ca2+ Channels Abolishes Depolarization-Induced Ca2+ Release in Arterial Smooth Muscle CIRCULATION RESEARCH

Proyectos de Investigación

Fecha de inicio Fecha de fin Rol Denominación Agencia financiadora
26/03/2013 31/03/2018 Investigador/a Canales de Calcio Tipo T en Cardiomiocitos: Regulación por la Hipoxia y por la Ruta Rho-Rock. (P11-CTS-7045) Junta de Andalucía - Consejería de Innovación, Ciencia y Empresas (Autonómico)
01/04/2009 31/03/2014 Investigador/a Nuevo Papel de los Canales de Ca2+ en el Control del Tono Vascular y su Relación con Mecanismos de Sensibilización a Ca2+ de la Contracción (P08-CTS-03530) Junta de Andalucía - Consejería de Innovación, Ciencia y Empresas (Autonómico)
01/01/2007 31/12/2012 Investigador/a Sensibilidad al oxígeno y neurodegeneración (CONFMB-001/2006) Fundación Marcelino Botín (Nacional)
El investigador no tiene ningún resultado de investigación asociado